A 63-year-old male suffers a myocardial infarction (heart attack) due to extensive coronary atherosclerosis. The myocardium is initially ischemic and then undergoes necrosis and is subsequently infiltrated by inflammatory cells. Once the inflammatory cells clear the necrotic tissue, cardiac fibroblasts are activated and replace the damaged tissue with a fibrotic scar. The scar is noncontractile, not depolarizable.
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